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Kategorien: Alle - pathogens - cytokines

von Ingrid Nathalia Tobar Tobar Vor 3 Jahren

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PERIODONTAL DISEASE

The progression of periodontal disease involves a series of phases, each marked by distinctive microbial activity and immune responses. Initially, bacteria accumulate, leading to an overgrowth of plaque, which is characteristic of early gingivitis.

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Biochemical mediators of the immune system

PERIODONTAL DISEASE

The progression of periodontal disease involves a series of phases, each marked by distinctive microbial activity and immune responses. Initially, bacteria accumulate, leading to an overgrowth of plaque, which is characteristic of early gingivitis.

Sputum culture

SolveSCI

Ankylosing Spondylitis1 (Erica Zeitz)

Innate immunity

.

Biochemical mediators of the immune system

MMPs

- Facilitate normal turnover of the connective tissue matrix - Collagen destruction

Prostaglandins

- Increase vascular permeability allowing inmune cells and complement to move to the infection site - PGE2 Alveolar bone destruction

Cytokines

- Recruit PMNs and macrophages to the infection site. - Initiate tissue destruction and bone

microbiome

bacteria

The presence of periodontal pathogens does not necessarily mean that an individual will experience periodontitis.

PERIODONTAL DISEASE

Pathogenesis

4. periodontitis - tissue destruction phase

3. Established gingivitis - subgingival plaque phase

2. Early gingivitis - plaque overgrowth phase

1. Early bacterial accumulation phase

Host response

Dysbiosis

Mindomo

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PERIODONTAL DISEASE

The progression of periodontal disease involves a series of phases, each marked by distinctive microbial activity and immune responses. Initially, bacteria accumulate, leading to an overgrowth of plaque, which is characteristic of early gingivitis.

Sputum culture

SolveSCI

Ankylosing Spondylitis1 (Erica Zeitz)

Innate immunity

.

Biochemical mediators of the immune system

MMPs

- Facilitate normal turnover of the connective tissue matrix - Collagen destruction

Prostaglandins

- Increase vascular permeability allowing inmune cells and complement to move to the infection site - PGE2 Alveolar bone destruction

Cytokines

- Recruit PMNs and macrophages to the infection site. - Initiate tissue destruction and bone

microbiome

bacteria

The presence of periodontal pathogens does not necessarily mean that an individual will experience periodontitis.

PERIODONTAL DISEASE

Pathogenesis

4. periodontitis - tissue destruction phase

3. Established gingivitis - subgingival plaque phase

2. Early gingivitis - plaque overgrowth phase

1. Early bacterial accumulation phase

Host response

Dysbiosis

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