genetic mutations were more susceptible
results in
Results in these phenotypes
Mutations arise due to the lack of the GBA enzyme

Gaucher Disease

Genetics

Mutations

Genotype

GBA gene is mutated

located on chromosome 1

there is a lack of the GBA enzyme

Responsible for breaking down glucosylceramide

A type of fat

Mutation occurs in both copies of the GBA gene

Changes the amino acid in the GBA enzyme

Which changes the structure of the enzyme

causing the enzyme to not function properly

Phenotype

Enlarged spleen

arthritis

enlarged liver

Anemia

decreased amount of red blood cells

Inheritance pattern

Autosomal Recessive

Affects both sexes equally

Two copies of the abnormal gene must be present

Both parents must be carriers

Genetic technologies

Genetic engineering

Enzyme replacement therapy

IV infusions

Prenatal tests

Chorionic villus sampling

Amniocentesis

Evolution

How mutations arose

GBA Enzyme

Genetic mutation

Lead to the lack of the GBA enzyme

leads to a lysosomal storage disease

Gaucher disease

inherited from parents

Patterns of evolution that lead to this

Mutations occurring inside the body

Founder effect

when a small portion of a large population establishes a new isolated community

Similarities to other organisms with this mutation

People with Parkinson's disease

GBA mutations cause a major genetic risk factor for getting Parkinson's disease

People with lysosomal storage disorders

Gaucher disease is a lysosomal storage disorders

people with these disorders will experience similar symptoms

Lysosomal storage disorders

inherited metabolic disorders

Cystinosis

Batten Disease

Fabry Disease

The genetic mutation of the GBA gene

mutations are inherited from the parents

leads to the reduction of genetic diversity

increasing the frequency and risk of a new disease in the community

lead to Jewish communities having a higher percentage of gaucher disease

because of their isolated community with similar alleles and genes

Have the same mutation as people with Gaucher disease

A decrease in the GBA enzyme

Similar gene mutations but different phenotypes

Speculation (but with some backed up information)