METALLEAD

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NETBALL

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Alcoholic Liver Disease

由Angela Matthews

Stroke

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AMIRA AND HALIDAH (INPUT)

由nur halidah

GROUP 1

Members: Jimmy Leon Jhon Romero Andres Coral Anthony Cardenas

METAL LEAD

Control a) Evaluation of the work environment and correction of deficiencies that condition the exposure to the contaminant. b) Worker education on hygiene and personal protection standards c) Quarterly control until normalization of the values.

Sequence of signs and symptoms associated with lead toxicity: Mild toxicity from lead exposure can cause: • slight fatigue • irritability • lethargy • occasional abdominal discomfort

HOLA

Characteristics: It is a bluish-gray, malleable and ductile metal, Whose melting point is at 327 ºC. Resistant to sulfuric acid, dissolves quickly in nitric acid and is solubilized by organic acids.

C.M.P.: 0,15 mg/m3 TLV-TWA: 0,05 mg/m3 (ACGIH)

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Uses and exposure:

Manufacture of accumulators

Pb and Zn metallurgy.

Lead and zinc mines.

Its main oxides are:

Litargirio (PbO)

Lead dioxide (PbO2)

Minium (Pb3O4)

Exam Biannually

Delta-amino-levulinic acid Value normal: <4.5 mg / g creatinine. Biological Exposure Index: 10 creat mg / g

Free protoporphyrins Normal value: <75 mcg / 100 ml red blood cells. Index Biological Exposure: up to 300 mcg / 100 ml red blood cells.

Lead value Normal value: <30 mcg / 100 ml of blood. Currently the ACGIH has Biological Exposure Index set in 30 mcg / 100 ml of blood.

Effects detection toxic ANNUALLY

PLASMA, CREATININ

BLOOD, UREA

HEMOGRAM

Toxicity Acute poisoning does not exist in industry.

Topic principal

Chronic exposure Lead targets are: a) S.N.C. and Peripheral b) Bone marrow c) Gonads d) Kidney