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Categories: All - pathogens - cytokines

by Ingrid Nathalia Tobar Tobar 3 years ago

388

PERIODONTAL DISEASE

The progression of periodontal disease involves a series of phases, each marked by distinctive microbial activity and immune responses. Initially, bacteria accumulate, leading to an overgrowth of plaque, which is characteristic of early gingivitis.

Multiple Sclerosis (MS) Pathophysiology

by Qabas Al-Jobori

Immunology

by Ravi Chacko

Rheumatoid Arthritis

by Anthony Traboulsi

Ankylosing Spondylitis1 (Erica Zeitz)

by Erica Zeitz

.

Biochemical mediators of the immune system

MMPs

PERIODONTAL DISEASE

The progression of periodontal disease involves a series of phases, each marked by distinctive microbial activity and immune responses. Initially, bacteria accumulate, leading to an overgrowth of plaque, which is characteristic of early gingivitis.

Multiple Sclerosis (MS) Pathophysiology

Immunology

Rheumatoid Arthritis

Ankylosing Spondylitis1 (Erica Zeitz)

.

Biochemical mediators of the immune system

MMPs

- Facilitate normal turnover of the connective tissue matrix - Collagen destruction

Prostaglandins

- Increase vascular permeability allowing inmune cells and complement to move to the infection site - PGE2 Alveolar bone destruction

Cytokines

- Recruit PMNs and macrophages to the infection site. - Initiate tissue destruction and bone

microbiome

bacteria

The presence of periodontal pathogens does not necessarily mean that an individual will experience periodontitis.

PERIODONTAL DISEASE

Pathogenesis

4. periodontitis - tissue destruction phase

3. Established gingivitis - subgingival plaque phase

2. Early gingivitis - plaque overgrowth phase

1. Early bacterial accumulation phase

Host response

Dysbiosis

Mindomo

More Info

Faq

PERIODONTAL DISEASE

The progression of periodontal disease involves a series of phases, each marked by distinctive microbial activity and immune responses. Initially, bacteria accumulate, leading to an overgrowth of plaque, which is characteristic of early gingivitis.

Multiple Sclerosis (MS) Pathophysiology

Immunology

Rheumatoid Arthritis

Ankylosing Spondylitis1 (Erica Zeitz)

.

Biochemical mediators of the immune system

MMPs

- Facilitate normal turnover of the connective tissue matrix - Collagen destruction

Prostaglandins

- Increase vascular permeability allowing inmune cells and complement to move to the infection site - PGE2 Alveolar bone destruction

Cytokines

- Recruit PMNs and macrophages to the infection site. - Initiate tissue destruction and bone

microbiome

bacteria

The presence of periodontal pathogens does not necessarily mean that an individual will experience periodontitis.

PERIODONTAL DISEASE

Pathogenesis

4. periodontitis - tissue destruction phase

3. Established gingivitis - subgingival plaque phase

2. Early gingivitis - plaque overgrowth phase

1. Early bacterial accumulation phase

Host response

Dysbiosis

Spread the word about Mindomo

Mindomo

More Info

Faq